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J9140 J9150 J9151 J9160 J9165 J9170 J9178 J9180 J9181 J9182 J9185 J9190 J9200 J9201 J9202 J9206 J9208 J9209 J9211 J9212 J9213 J9214 J9215 J9216 J9217 J9218 J9219 J9230 J9245 J9250 J9260 J9263 J9265 J9266 J9268 J9270 J9280 J9290 J9291 J9293 J9300 J9310 J9320 J9340 Dacarbazine 200 MG inj Daunorubicin, 10 mg Daunorubicin citrate liposome Daunoxome ; 10 mg Denileukin diftitox, 300 mcg Ontak ; Diethylstilbestrol diphosphate Stilphostrol ; 250 mg injection Docetaxel Taxotere ; 20 mg Inj, epirubicin hcl, 2 mg Epirubicin HCl injection 50 mg Ellence ; Etoposide 10 MG inj VePesid ; Etoposide 100 MG inj Fludarabine phosphate inj 50 mg Fludara ; Fluorouracil injection 500 mg Floxuridine injection 500 mg Gemcitabine HCl 200 mg Goserelin acetate implant Zoladex ; per 3.6 mg Irinotecan injection 20 mg Ifosfomide injection 1 Gm Mesna injection Mesnex ; 200 mg Idarubicin hcl 5 mg Interferon alfacon-1, recombinant, 1 mcg Interferon alfa-2a inj, 3 million units Interferon alfa-2b inj 1 million units Interferon alfa-n3 inj human leukocyte derived ; 250, 000 IU Interferon gamma 1-b inj, 3 million units Leuprolide acetate suspension Lupron Depot ; for depot suspension, 7.5 mg Leuprolide acetate injection Lupron ; per 1 mg Leuprolide acetate implant, 65 mg Mechlorethamine hcl inj nitrogen mustard ; , Mustargen 10 mg Inj melphalan hcl 50 MG Methotrexate sodium inj 5 mg Methotrexate sodium inj 50 mg Oxaliplatin Paclitaxel injection 30 mg Pegaspargase single dose vial Pentostatin injection 10 mg Plicamycin Mithracin ; inj 2.5 mg Mitomycin 5 MG inj Mitomycin 20 MG inj Mitomycin 40 MG inj Mitoxantrone hcl 5 MG Genetuzumab ozogamicin, 5 mg. Rituximab 100 mg Streptozocin 1 GM Thiotepa 15 mg Page 16. During 1998, exchange rate fluctuations had no significant impact on our net sales, due principally to the overall stability of the value of the dollar against the french franc.

Patients who were previously refractory to fludarabine did not benefit substantially from the addition of oblimersen, and because there were so many refractory patients in both arms of the trial, the survival curves were not significantly different between the groups, o' brien says. 1. Hamblin TJ. Autoimmune complications of chronic lymphocytic leukemia. Semin Oncol 2006; 33: 2309. Pritsch O, Maloum K, Dighiero G. Basic biology of autoimmune phenomena in chronic lymphocytic leukemia. Semin Oncol 1998; 25: 3441. Sthoeger ZM, Wakai M, Tse DB, et al. Production of autoantibodies by CD5-expressing B lymphocytes from patients with chronic lymphocytic leukemia. J Exp Med 1989; 169: 25568. Hall A, Vickers MA, McLeod E, Barker RN. Rh autoantigen presentation to helper T cells in chronic lymphocytic leukemia by malignant B cells. Blood 2005; 105: 200715. Sthoeger Z, Sthoeger D, Shtalrid M, Sigler E, Geltner D, Berrebi A. Mechanism of autoimmune hemolytic anemia in chronic lymphocytic leukemia. J Hematol 1993; 43: 25964. Centola M, Lin K, Sutton C, et al. Production of anti-erythrocyte antibodies by leukemic and nonleukemic B cells in chronic lymphocytic leukemia patients. Leuk Lymphoma 1996; 20: 4659. Stevenson FK, Hamblin TJ, Stevenson GT, Tutt AL. Extracellular idiotypic immunoglobulin arising from human leukemic B lymphocytes. J Exp Med 1980; 152: 148496. Tarlinton D, Stall AM, Herzenberg LA. Repetitive usage of immunoglobulin VH and D gene segments in CD5 + Ly-1 B clones of NZB x NZW ; F1 mice. EMBO J 1988; 7: 370510. Borche L, Lim A, Binet JL, Dighiero J. Evidence that chronic lymphocytic leukemia B lymphocytes are frequently committed to production of natural autoantibodies. Blood 1990; 76: 5629. Marcus H, Shimoni A, Ergas D, et al. Human mouse radiation chimera generated from PBMC of B chronic lymphocytic leukemia patients with autoimmune hemolytic anemia produce anti-human red cell antibodies. Leukemia 1997; 11: 68793. Beaume A, Brizard A, Dreyfus B, Preud'homme JL. High incidence of serum monoclonal Igs detected by a sensitive immunoblotting technique in B-cell chronic lymphocytic leukemia. Blood 1994; 84: 121619. Dighiero G. Relevance of murine models in elucidating the origin of B-CLL lymphocytes and related immune-associated phenomena. Semin Hematol. 1987; 24: 24051. Myint H, Copplestone JA, Orchard J, et al. Fludarabine-related autoimmune haemolytic anaemia in patients with chronic lymphocytic leukaemia. Br J Haematol. 1995; 91: 3414. Eichhorst BF, Busch R, Hopfinger G, et al. Fludarabine plus cyclophosphamide induces higher remission rates and longer progression free survival than fludarabine alone in first line therapy of advanced CLL: results of a phase III Study CLL4 Protocol ; of the German CLL Study Group. 45th ASH Meeting, San Diego, California. Blood 2003; 102: 243a. Robak T, Blasinska-Morawiec M, Krykowski E, Hellmann A, Konopka L. Autoimmune haemolytic anaemia in patients with chronic lymphocytic leukaemia treated with 2-chlorodeoxyadenosine cladribine ; . Eur J Haematol. 1997; 58: 10913. Catovsky D, Richards S. Incidence of hemolytic anemia after chemotherapy in the CLL4 trial. A possible protective role for fludarabine plus cyclophosphamide. 46th ASH Meeting, San Diego, California. Blood 2004; 104: 480a. Shvidel L, Shtalrid M, Berrebi A. Intractable autoimmune hemo.

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Table 1. Staining intensity of monoclonal antibodies in immunofluorescence and immunoperoxidase methods 1, 4-butanediol, supplement warning #195 p.156 AA oxidopathy #186 p.92-102 AA oxidopathy, arrested growth in children #201 p.52-62 AAACa active absorbable algal calcium ; , bioavailability #229 p.110-14 Abandonment, homeopathy #239 p.152 + AbioCor, artificial heart #209 p.14 Abscopal antitumor effect #238 p.37 Abstracts, Haworth Press #228 p.116 Abstracts, misinformation #197 p.34 ABVA-1 purified phosphatidylcholine ; , immune function #225 p.52-55 Academic decline, nutrition #201 p.108-11 Academic performance, gender #205 p.106 + Access to Medical Treatment Act #196 p.134 + , #199 p.126 + Accidents, homeopathy #204 p.42-51 Acetaminophen, asthma #211 p.159 Acetyl-L-Carnitine, neuron-brain effects #229 p.125-26 Acetylcholinesterase inhibitor, sage oil #202 p.151 Achlorhydria, inositol hexaniacinate #235 p.72-5, #238 p.70-71 Acidosis, enzyme therapy #233 p.90-94 Acne, DHEA #225 p.30 Acromelic gangrene, Chinese medicine #241 p.48-51 Acupressure #186 p.36-37 Acupressure, back injury #201 p.130-31 Acupressure, cold prevention #233 p.130-31 Acupressure, ear #192 p.124-25 Acupressure, heart #191 p.20-21 Acupressure, heart attacks #209 p.33-34 Acupressure, home care #189 p.40-41 Acupressure, indigestion #215 p.150-51 Acupressure, low blood sugar #205 p.34-35 Acupressure, lower back pain #208 p.32-33 Acupressure, neck shoulder pain #199 p.38-39 Acupressure, pain relief #190 p.22-23 Acupressure, self-care #202 p.140-41 Acupressure, sore muscles #210 p.134-35 Acupuncture, anxiety treatment #205 p.96 Acupuncture, asthma respiratory illness #245 p.127-28 Acupuncture, California training law #233 p.21 Acupuncture, Cuba #226 p.22-24 Acupuncture, dizziness vertigo #244 p.146-47 Acupuncture, drug detoxification #216 p.78-80 + Acupuncture, Five Element #201 p.116-25 Acupuncture, hypertension #229 p.60-66 Acupuncture, low back pain study #187 p.37 Acupuncture, migraines #187 p.12-13 Acupuncture, osteoporosis #238 p.133 Acupuncture, piriformis syndrome #240 p.136-37 Acupuncture, senile dementia #228 p.38-42 Acupuncture, sports injuries #240 p.68-70 Acupuncture, Tai Sophia Institute community program #216 p.84-86 Acupuncture, warm needle #234 p.136-37 Acupuncture-moxibustion, AHDH #243 p.48-50 Acupuncture degree, University of Bridgeport Connecticut ; #193 p.56 Acupuncture Stimulator #207 p.101 Acute myocardial infarction, intravenous magnesium #237 p.114 Adams, Patch, MD #187 p.77-78 Adaptation, humans & diet #228 p.126 Adaptogens, adrenal function #220 p.65-68 Addiction, aspartame #198 p.52-57 and flumist.
Fludarabine and Campath-1H are both approved for the treatment of CLL but produce significant cellular immune suppression. To assess the impact of KRN5500 on normal T cells and B cells, these were isolated from healthy volunteers and incubated in KRN5500 for 4 hours followed by incubation in fresh medium until 96 hours. As shown in Figure 1, the viability of both normal T cells and B cells exceeded 50% of the media control in all patients at the 1 M or less concentration. In contrast, the median LC50 for the CLL patients studied was 0.209 M, suggesting KRN5500 demonstrates some selectivity for the malignant B cell. In an attempt to determine if the cytotoxicity induced by KRN5500 was due to an increase in apoptosis, mononuclear cells from 5 CLL patients were incubated in medium alone or 0.134 M or 1.34 M KRN5500 for 24 hours. At this point, cleavage of caspase-3 and PARP, which serves as a substrate for this activated effector caspase, was assessed. Figure 2A demonstrates one such representative patient demonstrating a dose-dependent increase in active 29 kDa heterodimer of caspase-3 as assessed by flow cytometry at 0.134 M and 1.34 M concentration of KRN5500. Utilizing annexin VPI staining, we demonstrated similar findings at the KRN5500 concentrations of 0.134 M or 1.34 M data not shown ; , but apoptosis was completely abrogated by addition of.

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The successful clinical use of freeze-dried allogeneic rotator cuff to repair massive chronic ruptures of the rotator cuff of the shoulder. In their patients there was no graft rejection, while improved movement and relief of pain were usually attained. At re-operation the graft was found to be completely reorganised as a viable rotator cuff. We did not use freeze-drying because of the. Wyatt, L. S., P. L. Earl, L. A. Eller, and B. Moss. 2004. Highly attenuated smallpox vaccine protects mice with and without immune deficiencies against pathogenic vaccinia virus challenge. Proc Natl Acad Sci U S A 101: 4590-5 and fluphenazine. The significant improvements in off-period contralateral bradykinesia at one week 36 percent change; 95 percent confidence interval, 23 to 49 percent ; and rigidity 51 percent change; 95 percent confidence interval, 32 to 68 percent ; were sustained in both the one-year and the two-year groups. Improvement in tremor was sustained in the oneyear group 53 percent change; 95 percent confidence interval, 35 to 71 percent ; . There was a tendency toward sustained improvement in the two-year group, but the changes in the scores were not statistically significant, because of the correction for multiple comparisons and the small number of patients 10 ; with tremor in this group. Significant improvement in the off-period composite score for postural instability and gait disorder was lost between 6 and 12 months in the one-year group and between 3 and 6 months in the two-year group. Improvement in off-period ipsilateral bradykinesia was not sustained for more than three months in either group, and changes in the scores for other ipsilateral symptoms were not significant. As seen in the group of 39 patients at six months Table 2 ; , changes in the on-period scores were generally not significant or were unsustained Diagnosed with cll n 18 ; and failed fludarabine within 6 months of therapy, or pll n 5 ; and failed 1 previous therapy and flurazepam.
Findings are only restrictedly transferable. Resistance markers are not routinely assessed in subSaharan Africa which is unlikely to change soon. Nevertheless, for epidemiological purposes we consider peripheral blood dhfr genotyping both useful and sufficiently precise to give an estimate of SP-resistance in parasites infecting pregnant women. This should allow to examine the dynamics of SP-resistance in infections occurring despite IPT. Once the predictive value of the dhfr mutations for IPT with SP has been established, monitoring these markers will provide.

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Notochord C4S Ref. 51 u, chondroitin. The inhibition capacity of oligosaccharide Fractions I and II from CS-2a were either similar or only marginally lower compared with those from CS-2b not shown ; . Inhibition of IRBC binding to BCSPG-2a were similar to that observed in the case of BCSPG-2b, except that a 10-20% higher inhibition was observed for each inhibitor used. Note: The inhibitory capacity of the 6-mer prepared by the testicular hyaluronidase digestion of C4S with 36% 4-sulfated disaccharide was similar to that by the intact C4S with 36% 4-sulfate and flurbiprofen.

Reintroduction of fludarabine for her relapsing disease induced autoimmune haemolytic anaemia. Dormant seeds of diploid barley, variety Himalaya C.I. 620 ; , were used in these experiments. Tests run in 1947 were performed with the stock orginally obtained from D. W. ROBERTSON. order to obtain as nearly In homozygous a line as possible for future studies, a single plant selection was increased in the summer of 1948. Since 1949 the tests have been made with the progeny from this single plant and fluvastatin. Nov 1, 2005 Waselenko JK, Burrows A, Lucas M, Ekstrand JR, Myhand R, Lee N, Edenfield WJ, Byrd JC. Low Dose Recombinent Interleukin-2 rIL-2 ; Following High Dose Chemotherapy HDC ; and Autologous Peripheral Blood Stem Cell Transplantion PBSCT ; in Patients with Chronic Lymphocytic Leukemia CLL ; , Mantle Cell MCL ; , and Follicular Lymphoma FL ; . Blood. 96: 350b 2000. Marcucci G, Bloomfield CD, Balcerzak SP, Kourlas PJ, Stanley HR, Fingert H, Mahraby EA, Lucas D, Chen KK, Byrd JC, Kraut EH, Grever MR, Caligiuri MA. Phase I Trial of Genasense G3139, Genta, Inc. ; , a BCL-2 Antisense AS ; , in Refractory REF ; or Relapsed REL ; Acute Leukemia AL ; . Blood . 96: 119a, 2000. Bloomfield CD, Byrd JC, Farag SS, Mrozek K, Dodge RK, Archer KJ, Whitman S, Larson RA, Carroll AJ, Caligiuri MA. Cytogenetics for Treatment Stratification in Adult Acute Myeloid Leukemia AML ; . Annals of Hematology. 80: S37, 2001. Marcucci G, Bloomfield CD, Balcerzak SP, Fingert H, Maghraby EA, Lucas D, Chen KK, Byrd JC, Kraut EH, Grever MR, Caligiuri MA. Phase I Trial of Genasense G3139, Genta, Inc. ; , a BCL-2 Antisense AS ; , in Refractory REF ; or Relapsed REL ; Acute Leukemia AL ; . American Society of Clinical Oncology. 20: 1149, 2001. Link BK, Wang H, Byrd JC, Leonard JP, Davis T, Flinn I, Hall WC, Turner JF, Bowles J, Shannon M, Levitt D, Weiner GJ, Prolonged Cilnical Responses in Patients with Follicular Lymphoma Treated on a Phase I Trial of the Anti-HLA-DR Monoclonal Antibody Remitogen Hu1D10 ; . Blood. submitted ; Maghraby E, Murphy T, Parthun M, Klisovic M, Sklenar A, Archer K, Whitman S, Grever M, Byrd JC, Marcucci G. Depsipeptide FR901228 ; Induces Lysine-Specific Histone Acetylation, Differentiation and Apoptosis is Acute Myeloid Leukemia Cells and Demonstrates Synergy with Decitabine. Blood. 98: 11 103a, Byrd JC, Mrozek K, Dodge R, Carroll AJ, Edwards C, Pettenati MJ, Patil SR, Larson RA, Bloomfield C. Pre-treatment Cytogenetics Predict Initial Induction Success and Overall Survival in Adult Patients with De Novo Acute Myeloid Leukemia: Results From CALGB 8461. Blood. 98: 11 457a, Link, BK, Kahl B, Czuczman M, Powell BL, Leonard JP, Ansell S, Porcu P, Byrd JC, Lazarus HM, Flinn I, Jones GJ, Levitt D, Hall W, Weiner GJ. A Phase II Study of Remitogen Hu1D10 ; , a Humanized Monoclonal Antibody in Patients with Relapsed or Refractory Follicular, Small Lymphocytic, or Marginal Zone MALT B-Cell Lymphoma. Blood. 98: 11 606a, Byrd JC, Peterson B, Piro L, Saven A, Vardiman JW, Larson RA, Shiffer CA, Cladribine has Clinical Activity in Patients with Fludarabine Refractory Chronic Lymphocytic Leukemia Who Lack Pre-Treatment Cytopenias: Results From CALGB Study 9211. Blood. 98: 11 634a, Lucas M, Flinn IW, Goodrich AL, Murphy T, Park K, Grever MR, Lehman T, Byrd JC. TNF- and TNF- Promotor Polymorphisms May Relate to Response to Rituximab Therapy. Blood. 98: 11 634a, Byrd JC, Peterson BL, Park K, Morrison VL, Vardiman JW, Jacobson R, Rai K, Larson RA. Concurrent Rituxumab and Fludarabine has a Higher Complete Response Rate Than Sequential Treatment in Untreated Chronic Lymphocytic Leukemia CLL ; Patients: Results From CALGB 9712. Blood. 98: 11 772a and fludarabine.

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Fludarabine phosphate as a single agent, or in combinations such as fludarabine, mitoxantrone, dexamethasone FMD ; may yield beneficial responses in relapsed disease. In several studies, response rates with fludarabine range from 32%62%. FMD has been reported to induce 90% response rates, and 47% CRs, in patients with recurrent lymphoma and a good prognosis. Purine analogues are also associated with significant immunosuppression and focalin.
Combination treatments B-CLL cells were cultured at a density of 5 X 106 cells ml in RPMI 160 containing 10% FBS and treated with the cell permeable form of fludarabine fludarabine des-phosphate ; at concentrations from 0.2 to 20 M, cladribine 2-CdA ; at concentrations from 1 to 100. Of combination chemotherapy with vincristine and prednisone. Although the treatment proved to be very effective in the bone marrow, leukemia relapse occurred in the central nervous system. Intrathecal chemotherapy consisting of MTX 15mg ; , cytarabine 40mg ; , and dexamethasone 4mg ; was administered a total of 5 times. Prior to the conditioning regimen, 12Gy of whole brain irradiation was performed. The conditioning regimen consisted of TBI and CY, followed by bone marrow transplantation BMT ; from an HLA-matched unrelated donor. Tacrolimus and MMF were used for GVHD prophylaxis. 2.7 x 108 cells kg of nucleated cells with 2.5 x 106 cells kg of CD34 + cells were infused. On day + 8, the patient experienced right upper quadrant abdominal pain and tenderness and showed a weight gain of 6.8% accompanied by renal dysfunction. Ultrasonography showed hepatomegaly, enlarged portal vein diameter and ascites. Although total bilirubin was 1.3mg dl, a diagnosis of SOS was established, and defibrotide at a dose of 1200mg was started in combination with 600mg of UDCA. On day + 13 renal dysfunction and liver dysfunction began to improve, neutrophil engraftment was achieved on day + 18 and on day + 22, complete response to defibrotide was achieved. From day + 24, defibrotide was tapered off without any deterioration in the patient's overall condition. As intrathecal chemotherapy was performed repeatedly after BMT, platelet engraftment was achieved later. Three months after the onset of SOS the patient is in good clinical condition without any liver dysfunction or relapse of CML. Data were summarized from the retrospective review of the medical charts and laboratory findings for four patients who received defibrotide for treatment of SOS after HSCT at our hospital. Patient characteristics. Patient age ranged from 34 to 59 years with a median age of 54 years Table 1 ; . All four patients showed evidence of underlying hematological malignancy, one underwent autologous stem cell transplantation SCT ; and others allogeneic SCT, two of whom underwent CBT. The patient undergoing autologous SCT received MCVC, and those treated with allogeneic SCT except case 3 underwent TBI and CY. Patient 3 had previously been administered a preparative regimen of TBI and CY. Subsequently the patient was treated with reduced-intensity stem cell transplantation RIST ; consisting of fludarabine and busulfan. Several different GVHD prophylaxis regimens were used. Clinical features. All patients met the Seattle criteria for SOS Table 2 ; . Median time of onset of SOS was on day + 12 range, 7 19 ; , and the median bilirubin at the onset of SOS was 2.6mg dl range, 1.3 3.0 ; . Patient condition and Outcome. All patients achieved neutrophil engraftment, and three platelet engraftment Table 3 ; . All of them suffered from renal dysfunction, and two cases of severe SOS with pulmonary dysfunction had a fatal outcome Table 4 ; . The administered daily dose of defibrotide varied widely, while none of the patients had to discontinue defibrotide administration because of significant adverse effects. CR was clinically observed in two patients. Case 3 died of acute GVHD, TMA of ileum, and CMV infection, but not of SOS and case 1 of SOS and CMV infection. However, a transient reduction in the latter's total bilirubin suggested a certain effect of defibrotide on SOS. Thus, three of the four patients with SOS were considered to have responded to defibrotide therapy and follistim.

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