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This release contains statements that constitute forward-looking statements within the meaning of the U.S. Private Securities Litigation Reform Act of 1995. These statements are based on management's current expectations or beliefs and are subject to a number of factors and uncertainties that could cause actual results to differ materially from those described in the forward-looking statements. The following factors, among others that are described in our Form 20-F as filed with the US Securities and Exchange Commission on June 25, 2003 and in the Reference Document filed with the French Commission des Oprations de Bourse on April 23, 2003, could cause actual results to differ materially from those described in the forward-looking statements: the ability of Sanofi-Synthlabo to expand its presence profitably in the United States; the success of Sanofi-Synthlabo's research and development programs; the ability of Sanofi-Synthlabo to protect its intellectual property rights; and the risks associated with reimbursement of health care costs and pricing reforms, particularly in the United States and France. Sanofi-Synthlabo does not undertake any obligation to provide updates or to revise any forward-looking statements. Investors and security holders may obtain a free copy of the Form 20-F and any other documents filed by Sanofi-Synthlabo with the US Securities and Exchange Commission at sec.gov, as well as of the Reference Document filed with the French Commission des Oprations de Bourse at cob or directly from Sanofi-Synthlabo on the web site sanofi-synthelabo.
Fig. 8. A ; Effect of naloxone 5 mg kg ; on antinociception caused by. Studies done in Tropical Africa show an increase in parasitaemia prevalence with age suggesting immunity of the mothers who had been taking antimalarials before delivery can protect their babies in their 1st few months of life, which shows that this may only be true if the correct measures are taken to curb the frequency of bites by infected mosquitos. 50 infants aged between 1-12months were sampled during their clinic visits. Site of the study: Muhuru-Bay Health Center. Malaria rate of infection in the region: 80%. Laboratory profiles were done and 40 of them took 2 weekly prophylactics of antimalaria drugs, 2 months before delivery. 40 of the mothers were using untreated mosquito nets or not. Blood samples taken in the 1st month after delivery and others in their 2, 3, 4, & 6 months after delivery.
Group, but the sample in these studies were not large enough to assess the issue accurately. In our study, nearly half of all disease-progression events occurred in patients with a base-line CD4 cell count of less than 50 per cubic millimeter. The two most common primary disease-progression events were esophageal candidiasis and P. carinii pneumonia. The greater frequency of these events in the treatment-interruption group was not attributed to the rates of use of imidazoles or P. carinii prophylaxis. These results demonstrate the risks of treatment interruption and emphasize the importance of prophylaxis against opportunistic infections in patients with HIV infection in whom treatment is interrupted for any reason. This randomized study was designed specifically to assess the clinical outcome of a structured interruption of treatment. Although there were equal numbers of deaths in each group, significantly more primary disease-progression events occurred in the treatment-interruption group. Our results indicate that in patients with multidrug-resistant HIV infection, it is best to continue treatment with an optimized antiretroviral regimen and avoid the use of treatment interruption.

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For generations, handwashing with soap and water has been considered a measure of personal hygiene 1 ; . The concept of cleansing hands with an antiseptic agent probably emerged in the early 19th century. As early as 1822, a French pharmacist demonstrated that solutions containing chlorides of lime or soda could eradicate the foul odors associated with human corpses and that such solutions could be used as disinfectants and antiseptics 2 ; . In paper published in 1825, this pharmacist stated that physicians and other persons attending patients with contagious diseases would benefit from moistening their hands with a liquid chloride solution 2 ; . In 1846, Ignaz Semmelweis observed that women whose babies were delivered by students and physicians in the First Clinic at the General Hospital of Vienna consistently had a.
Universidad de Oriente We contributed with Bs. 53 million as down payment to commence the works to complete the Paseo Los Ilustres Sucrenses of this University. Santa Maria City School Bs. 276 million were donated to complete the classrooms of this school in Petare. Fitcar 2005 We contributed with the governmental initiative "Soap and Water for Caracas" whose objective was cleaning and rescuing the public spaces of the Capital under the joint action of the community, the mayoralties of the Metropolitan Area and the entities responsible for the maintenance and ornamentation of the city. To this initiative we contributed with Bs. 122.79 million. Toys for Christmas The Banesco employees and workers helped the Baby Jesus and Santa Claus to give out Christmas presents and moments of illusion by donating over 8, 400 toys, which represented a higher than Bs. 416 million investment. Oswaldo Guilln Foundation We support the Oswaldo Guilln Foundation whose objective is to assist institutions or projects working in favor of the health and education of the neediest children. Here we contributed with Bs. 10 million and naltrexone.

Mumps vaccine live ; . 684 Mupirocin.2065 Mupirocin calcium .2067 Mupirocinum.2065 Mupirocinum calcicum .2067 Musci medicati. 604 Mycobacteria 2.6.2. ; . 149 Mycophenolas mofetil.5.2-3239 Mycophenolate mofetil.5.2-3239 Mycoplasma gallisepticum vaccine inactivated ; .5.6-4491 Mycoplasmas 2.6.7. ; .5.8-5201 myo-Inositol .5.8-5325 myo-Inositolum.5.8-5325 Myristicae fragrantis aetheroleum . 2123 Myrrh .2069 Myrrha .2069 Myrrhae tinctura .2069 Myrrh tincture .2069 Myrtilli fructus recens. 1099 Myrtilli fructus siccus. 1099 Myxomatosis vaccine live ; for rabbits . 775 N Nabumetone .2073 Nabumetonum .2073 N-Acetyltryptophan.918 N-Acetyltryptophanum.918 N-Acetyltyrosine . 920 N-Acetyltyrosinum . 920 Nadolol . 2074 Nadololum. 2074 Nadroparin calcium .2075 Nadroparinum calcicum .2075 Naftidrofuryl hydrogen oxalate.2078 Naftidrofuryli hydrogenooxalas.2078 Nalidixic acid.2080 Naloxone hydrochloride dihydrate.5.7-5063 Naloxoni hydrochloridum dihydricum .5.7-5063 Naltrexone hydrochloride.5.1-2979 Naltrexoni hydrochloridum.5.1-2979 Nandrolone decanoate .5.5-4277 Nandroloni decanoas.5.5-4277 Naphazoline hydrochloride.2082 Naphazoline nitrate .5.3-3561 Naphazolini hydrochloridum .2082 Naphazolini nitras.5.3-3561 Naproxen.5.2-3245 Naproxen sodium .5.6-4643 Naproxenum.5.2-3245 Naproxenum natricum .5.6-4643 Narrow-leaved coneflower root .5.7-5064 Nasal drops and liquid nasal sprays.5.6-4473 Nasalia .5.6-4473 Nasal powders.5.6-4474 Nasal preparations .5.6-4473 Nasal preparations, semi-solid.5.6-4474 Nasal sprays liquid ; and nasal drops .5.6-4473 Nasal sticks.5.6-4474 Nasal washes .5.6-4474 Natrii acetas trihydricus . 2415 Natrii acetatis [1-11C] ; solutio iniectabilis .5.4-3885 Natrii alendronas . 2416 Natrii alginas . 2417 Natrii amidotrizoas . 2418 Natrii aminosalicylas dihydricus. 2419 Natrii ascorbas.5.6-4679 Natrii aurothiomalas.5.8-5363 Natrii benzoas. 2421 Natrii bromidum.2422 5422.

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McCoy et al. DN-TNF Inhibitor Rescues Nigral Dopamine Neurons a cyclin-dependent kinase 5-mediated pathway in a transgenic model of Alzheimer's disease. J Neurosci 25: 8843 8853. Leng A, Mura A, Feldon J, Ferger B 2005 ; Tumor necrosis factor-alpha receptor ablation in a chronic MPTP mouse model of Parkinson's disease. Neurosci Lett 375: 107111. Liu B, Du L, Hong JS 2000 ; Naloxone protects rat dopaminergic neurons against inflammatory damage through inhibition of microglia activation and superoxide generation. J Pharmacol Exp Ther 293: 607 617. Mabjeesh NJ, Frese M, Rauen T, Jeserich G, Kanner BI 1992 ; Neuronal and glial gamma-aminobutyric acid transporters are distinct proteins. FEBS Lett 299: 99 102. MacEwan DJ 2002 ; TNF receptor subtype signalling: differences and cellular consequences. Cell Signal 14: 477 492. Marchetti L, Klein M, Schlett K, Pfizenmaier K, Eisel UL 2004 ; Tumor necrosis factor TNF ; -mediated neuroprotection against glutamateinduced excitotoxicity is enhanced by N-methyl-D-aspartate receptor activation. Essential role of a TNF receptor 2-mediated phosphatidylinositol 3-kinase-dependent NF-kappa B pathway. J Biol Chem 279: 32869 32881. McGeer PL, Itagaki S, Boyes BE, McGeer EG 1988 ; Reactive microglia are positive for HLA-DR in the substantia nigra of Parkinson's and Alzheimer's disease brains. Neurology 38: 12851291. McGuire SO, Ling ZD, Lipton JW, Sortwell CE, Collier TJ, Carvey 2001 ; Tumor necrosis factor alpha is toxic to embryonic mesencephalic dopamine neurons. Exp Neurol 169: 219 230. Mitoma H, Horiuchi T, Hatta N, Tsukamoto H, Harashima S, Kikuchi Y, Otsuka J, Okamura S, Fujita S, Harada M 2005 ; Infliximab induces potent anti-inflammatory responses by outside-to-inside signals through transmembrane TNF-alpha. Gastroenterology 128: 376 392. Mogi M, Togari A, Tanaka K, Ogawa N, Ichinose H, Nagatsu T 1999 ; Increase in level of tumor necrosis factor TNF ; -alpha in 6-hydroxydopamine-lesioned striatum in rats without influence of systemic L-DOPA on the TNF-alpha induction. Neurosci Lett 268: 101104. Mogi M, Togari A, Kondo T, Mizuno Y, Komure O, Kuno S, Ichinose H, Nagatsu T 2000 ; Caspase activities and tumor necrosis factor receptor R1 p55 ; level are elevated in the substantia nigra from parkinsonian brain. J Neural Transm 107: 335341. Moore DJ, West AB, Dawson VL, Dawson TM 2005 ; Molecular pathophysiology of Parkinson's disease. Annu Rev Neurosci 28: 57 87. Nagatsu T, Sawada M 2005 ; Inflammatory process in Parkinson's disease: role for cytokines. Curr Pharm Des 11: 999 1016. Nishimura M, Mizuta I, Mizuta E, Yamasaki S, Ohta M, Kaji R, Kuno S 2001 ; Tumor necrosis factor gene polymorphisms in patients with sporadic Parkinson's disease. Neurosci Lett 311: 1 4. Oddo S, Caccamo A, Shepherd JD, Murphy MP, Golde TE, Kayed R, Metherate R, Mattson MP, Akbari Y, LaFerla FM 2003 ; Triple-transgenic model of Alzheimer's disease with plaques and tangles: intracellular Abeta and synaptic dysfunction. Neuron 39: 409 421. Olleros ML, Guler R, Vesin D, Parapanov R, Marchal G, Martinez-Soria E, Corazza N, Pache JC, Mueller C, Garcia I 2005 ; Contribution of transmembrane tumor necrosis factor to host defense against Mycobacterium bovis bacillus Calmette-Guerin and Mycobacterium tuberculosis infections. J Pathol 166: 1109 1120. Pasparakis M, Alexopoulou L, Episkopou V, Kollias G 1996 ; Immune and inflammatory responses in TNF alpha-deficient mice: a critical requirement for TNF alpha in the formation of primary B cell follicles, follicular dendritic cell networks and germinal centers, and in the maturation of the humoral immune response. J Exp Med 184: 13971411. Paxinos G, Watson C, Pennisi M, Topple A 1985 ; Bregma, lambda and the interaural midpoint in stereotaxic surgery with rats of different sex, strain and weight. J Neurosci Methods 13: 139 143. Pejovic V, Soskic V, Pan W, Kastin AJ 2004 ; Brain proteome of mice lacking the receptors for tumor necrosis factor alpha. Proteomics 4: 14611464. Przedborski S 2005 ; Pathogenesis of nigral cell death in Parkinson's disease. Parkinsonism Relat Disord 11 [Suppl 1]: S3S7. Przedborski S, Ischiropoulos H 2005 ; Reactive oxygen and nitrogen species: weapons of neuronal destruction in models of Parkinson's disease. Antioxid Redox Signal 7: 685 693. Ritter U, Meissner A, Ott J, Korner H 2003 ; Analysis of the maturation process of dendritic cells deficient for TNF and lymphotoxin-alpha reveals an essential role for TNF. J Leukoc Biol 74: 216 222 and namenda.

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G. ALLERGENIC SERUMS 1. All allergenic serums are covered as a medical benefit when pescribed by a primary care provider or his her designated referral provider. H. SUBSTANCE ABUSE DETERRENTS FORMULARY AGENTS COST DAY RANGE: $ 0.50 - $$ 2.50 3.00 - $$$ 5.00 15.00 methadone disulfuram naltrexone buprenorphine naloxone NOTE: METHADONE * ANTABUSE REVIA * SUBOXONE. Because of the reinforcing opioid effects of buprenorphine and its abuse potential in both dependent and nondependent opioid users, Suboxone and Subutex are registered in the United States as Schedule III narcotics under the Controlled Substances Act. 4.1.1 Physical dependence liability The physical dependence liability of buprenorphine has been evaluated in animals and is markedly lower than that of full mu receptor agonists.27, 28, 119, 120, Nevertheless, prolonged administration of high doses including those used in the treatment of opioid dependence ; can lead to physical dependence and symptoms of withdrawal following abrupt cessation of buprenorphine or challenge with very high doses of an opioid antagonist. The withdrawal syndrome following the abrupt discontinuation of buprenorphine is typically milder than seen with full agonists and may be delayed in onset.123 4.1.2 Abuse liability The effects of buprenorphine when administered sublingually and by injection have been extensively evaluated. As with all potent opiate agonists, parenteral abuse and illicit diversion have been reported worldwide.124, 125, 126, 127 In general, buprenorphine produces morphine-like opioid agonist effects that reach a ceiling effect at high doses when taken by these routes. In opioid-dependent subjects, IV and IM injections of buprenorphine-naloxone combinations have produced marked opioid-antagonist effects that precipitated an intense and rapid withdrawal syndrome.128, 129 The agonist and antagonist effects of buprenorphine and naloxone, alone and in combination, have undergone extensive examination in various populations: those dependent on heroin; those stabilized on morphine, methadone, hydromorphone, or buprenorphine; and nondependent, opioid-experienced subjects. Section 2.4 contains an in-depth report on pharmacodynamic responses of buprenorphine including subjective and physiological effects ; and buprenorphine naloxone challenges in various patient populations. 4.1.3 Cognitive impairment Suboxone and Subutex may impair the mental or physical abilities required for the performance of potentially dangerous tasks such as driving a car or operating machinery, especially during induction and dose adjustment. Patients should be cautioned about operating hazardous machinery, including automobiles, until they are reasonably certain that buprenorphine therapy is not adversely affecting their ability to engage in such activities. The effects of established weekly doses of buprenorphine 35 mg-210 mg ; , methadone 140 mg455 mg ; , and LAAM 199 mg-630 mg ; on simulated driving were compared with a control group of aged-matched, nondrug-using participants, with and without alcohol 0.7 mL kg and naratriptan.

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Just mailed off a letter to your editors, ref: fuel water, then read through the article about what is known not known concerning the cause of Joel Smith's fatal accident. Reminded me of one of my own personal "experiences." If there is a "moral" to my story, guess it would boil down to this: regardless of who is "in command, " there comes a time when you simply have to take command of the f --g machine now. The attachment is an excerpt from my life narrative as an Air Force fighter and test pilot, entitled Throw A Nickel On The Grass. As I've told your editors, you have an excellent magazine that consistently provides valuable information to Mooney Drivers. Enjoy.
No. Running a water line to the roof is the major cost, and that is peanuts usually hundreds of dollars, not thousands ; if done during initial construction. While you are at it, a 1" line costs little more than a 1 2" line, so run a 1" line with a 3 4" hose bib. Hose bibs on the roof save a lot of labor over the alternative: running hoses up the side of the building. And the trickle that you get from a long hose run doesn't compare with what you can do with a real plumbing riser and narcan Another derivative of oxymorphone is the narcotic antagonist naloxone narcan. About SUBOXONE Approved by Health Canada in May, 2007ix, SUBOXONE is indicated for substitution treatment of opioid drug dependence in adults. The intention of the naloxone component is to deter intravenous misuse. Patients prescribed SUBOXONE should be carefully monitored within a framework of medical, social and psychological support as part of a comprehensive opioid dependence treatment program.iv The approval is based on results of a four-week safety efficacy study in 326 subjects and nardil Progression of diabetic renal disease differ? Journal of Hypertension 13, S95S101. Benjamin, D., Grant, E. R. and Pohorecky, L. A. 1993 ; Naltrexone reverses ethanol-induced dopamine release in the nucleus accumbens in awake, freely moving rats. Brain Research 621, 137140. Birkenhager, T. K., Moleman, P. and Nolen, W. A. 1995 ; Benzodiazepines for depression? A review of the literature. International Clinical Psychopharmacology 10, 181195. Chalmers, J. 1993 ; The place of combination therapy in the treatment of hypertension. Clinical and Experimental Hypertension 15, 12991313. Cowen, M. S., Rezvani, A. H., Jarrott, B. and Lawerence, A. J. 1999 ; Ethanol consumption by Fawn-Hooded rats following abstinence: effects of naltrexone and changes in -opioid receptor density. Alcoholism: Clinical and Experimental Research 23, 10081014. Devine, D. P., Leone, P. and Wise, R. A. 1993 ; Mesolimbic dopamine neurotransmission by administration of -opioid receptor antagonists. European Journal of Pharmacology 24, 5564. DiChiara, G. and Imperato, A. 1988 ; Drugs abused by humans preferentially increase synaptic dopamine concentrations in the mesolimbic system of freely moving rats. Proceeding of the National Academy of Sciences of the USA 85, 52745278. Farren, C. K., Catapano, D. and O'Malley, S. 1997 ; Sertraline with naltrexone versus naltrexone alone in the treatment of alcohol dependence. Alcoholism: Clinical and Experimental Research 21 suppl. ; , 64 Abstract no. 366 ; . Froehlich, J. C. and Li, T.-K. 1993 ; Recent developments in alcoholism: opioid peptides. Recent Developments in Alcoholism 11, 187205. Froehlich, J. C., Harts, J., Lumeng, L. and Li, T.-K. 1990 ; Naloxone attenuates voluntary ethanol intake in rats selectively bred for high ethanol preference. Pharmacology, Biochemistry and Behavior 35, 385390. Gardell, L. R., Whalen, C. A., Chattophadyay, S., Cavallaro, C. A., Hubbell, C. L. and Reid, L. D. 1997 ; Combination of naltrexone and fluoxetine on rats' propensity to take alcoholic beverage. Alcoholism: Clinical and Experimental Research 21, 14351439. Grant, K. A. 1995 ; The role of 5-HT3 receptors in drug dependence. Drug and Alcohol Dependence 38, 155171. Hodge, C. A., Haraguchi, M., Erickson, H. L. and Samson, H. H. 1993a ; Ventral tegmental microinjections of quinpirole decrease ethanol and sucrose-reinforced responding. Alcoholism: Clinical and Experimental Research 17, 370375. Hodge, C. A., Samson, H. H., Lewis, R. S. and Erickson, H. L. 1993b ; Specific decreases in ethanol -- but not water-reinforced responding produced by the 5-HT3 antagonist ICS 205930. Alcohol 10, 191196. Koob, G. F., Rassnick, S., Heinrichs, S. and Weiss, F. 1994 ; Alcohol, the reward system and dependence. EXS 71, 103114. Lankford, M. F. and Myers, R. D. 1996 ; Opiate and 5-HT2a receptors in alcohol drinking: preference in HAD rats is inhibited by combination treatment with naltrexone and amperozide. Alcohol 13, 5357. Le, A. D. and Sellers, E. M. 1994 ; Interaction between opiate and 5-HT3 receptor antagonists in the regulation of alcohol intake. Alcohol and Alcoholism 29 suppl. 2 ; , 545549. Levine, J. H., Ferdinand, K. C., Cargo, P., Laine, H. and Lefkowitz, M. 1995 ; Additive effects of verapamil and enalapril in the treatment of mild to moderate hypertension. American Journal of Hypertension 8, 494499. Li, T.-K. and McBride, W. J. 1995 ; Pharmacogenetic models of alcoholism. Clinical Neuroscience 3, 182185. Mason, G. A., Rezvani, A. H., Grady, D. R. and Garbutt, J. C. 1994 ; The subchronic effects of the TRH analog TA-0910 and bromocriptine on alcohol preference in alcohol-preferring rats: development of tolerance and cross-tolerance. Alcoholism: Clinical and Experimental Research 18, 11961201. Mason, G. A., Rezvani, A. H., Overstreet, D. H. and Garbutt, J. C. 1996 ; The thyrotropin releasing hormone analog TA-0910 reduces voluntary alcohol intake of P rats subchronically in a limited scheduled access paradigm. Alcoholism: Clinical and Experimental Research 20, 10001003. Mason, G. A., Rezvani, A. H., Overstreet, D. H., Hamedi, M., Walker, C. H., Yang, Y. and Garbutt, J. C. 1997 ; Involvement of dopamine D2 receptors in the suppressive effect of the thyrotropin releasing hormone analog TA-0910 on alcohol intake in P rats. Alcoholism: Clinical and Experimental Research 21, 16231629.

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Clendeninn NJ, Petraitis M, Simon EJ, 1976. Ontological development of opiate receptors in rodent brain. Brain Res 118: 157-60 Coyle JT, Pert CB, 1976. Ontogenetic development of [3H1-naloxone binding in rat brain. Neuropharmacology 5: 555-60 Foster DL, Yellon SM, Olster DH, 1985. Internal and external determinants of the timing of puberty in the female. J Reprod Fertil 75: 327-44 Ho CL, Hammonds kG, Li CH, 1985. Opiate receptor binding profile in the rabbit cerebellum and brain membranes. Biochem Pharmacol 34: 925-3 1 leiri T, Chen HT, Meites J, 1979. Effects of morphine and naloxone on serum levels of LH and prolactin in prepubertal male and female rats. Neuroendocrinology 29: 288-92 Jacobson W, Wilkinson M, 1984. Opiate l3Hl-naloxone ; binding to hypothalamus and Res Bull 13: 481-85 Jacobson cerebral cortical slices of mouse brain. Brain and natalizumab. A. Pulse Oximetry Assessment Pulse Oximetry is not without limits and must NOT be used to supersede other assessments. The Fire Fighter Paramedic shall treat the patient and NOT the pulse oximeter's display. The patient's other key signs and symptoms must be assessed and evaluated so that the oximeter's readings are interpreted within the context of the patient's overall condition. The percentage of oxygen saturation measured by an oximeter only reflects the supplied pulmonary oxygenation and is not an indicator or measure of cellular oxygenation. Furthermore, it is useful both in the assessment of the patient and as an adjunct for evaluating the effectiveness of the airway management, ventilation, and oxygen enrichment provided. Oxygen saturation pressure SpO2 ; is a different measurement than the partial pressure of oxygen PaO2 ; which is commonly measured by laboratory blood gas analysis. Pulse Oximetry should be deferred until more urgent assessment and care priorities have first been resolved. Pulse oximetry is a diagnostic tool that, along with the patient's vital signs, chief complaint, mental status, and other considerations, may assist us in determining the patient's respiratory status. The pulse rate determined by the pulse oximeter is not an accurate indicator of the patient's pulse rate. Falsely low readings may occur in the following: a. patients with cold extremities or hypothermic patients b. patients with hemoglobin abnormalities c. patients without a pulse d. hypovolemic patients e. hypotensive patients Falsely normal or high oxygen saturation readings may occur in the following patients: a. anemic patients, carbon monoxide poisoning b. cyanide toxicity which is being treated with the antidote c. very bright lighting direct sunlight or nearby strong lamp ; Other factors affecting accurate readings: a. patient movement b. action of vasopressor drug c. peripheral vascular disease d. elevated bilirubin levels e. abnormal hemoglobin values f. IV diagnostic die has been administered in the last 24 hours and naloxone.

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Effects of opioid analgesics must be monitored carefully, because naloxone is short acting. In contrast, nalmefene, an opioid antagonist with a longer duration of action, has shown promise. Studies using ["C]carfentanil a radiotracer for u opiate receptors ; compared the duration of binding of naloxone with that of nalmefene and showed that naloxone and nalmefene have clearance half-times of 2 and 28 h, respectively. This study was conducted with a coincidence probe detector rather than a PET scanner 48 ; . From this study, it was concluded that nalmefene's longer blockade of opioid receptors is advantageous in the clinical reversal of narcotic anesthesia and opioid side effects and the reversal of opioid overdose and natrecor.
Naloxone is a pure antagonist, also derived from the opium poppy, and it will for sure send a person dependent on pure agonists into withdrawal. Active Member - Any pharmacist who has substantial professional activities in the area of infectious diseases pharmacotherapy or research may participate as a voting, active member of The Society. Prospective members must have been practicing or performing research in infectious diseases pharmacotherapy for at least two years after receipt of the terminal academic degree. Active member applicants must submit 2 letters of reference from fellow health care professionals attesting to substantial professional activities in the area of infectious disease pharmacotherapy or research and a current curriculum vitae. Associate Member - Pharmacist or non-pharmacist not meeting the requirements for the Active membership, but with an interest in the area of infectious disease pharmacotherapy, may participate as a non-voting member of The Society. Associate member applicants must submit 1 letter of reference from a fellow health care professional attesting to his her interest in the area of infectious disease pharmacotherapy or research along with a current curriculum vitae. Trainee-Associate Member - Pharmacist in either a residency or fellowship program with emphasis on infectious disease pharmacotherapy, and not more than two years past the receipt of the terminal degree, or student in an accredited school of pharmacy pursuing a degree in pharmacy, may participate as a non-voting member of The Society. Those individuals more than two years past the terminal degree should apply for active or associate status, whichever is appropriate. Trainee-Associate member applicants must provide a letter from their program director and student applicants must provide a letter from a professor. All applicants must also provide a current curriculum vitae and navane Indications: Moderate to severe pain Dosage: IV 1 mg, or 3 4 mg IM. 2 mg Naloxone should be available ; Contraindications: Smaller doses for older adults, temperature light sensitive and naltrexone.

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LEANDER, J. D.: Buprenorphine is a potent k-opioid receptor antagonist in pigeons and mice. Eur. J. Pharmacol. 151: 457-461, 1988. LEWIS, J. W.: Buprenorphine. Drug Alcohol Depend. 14: 363-372, 1985. LIGUORI, A., MORSE, W. H. AND BERGMAN, J.: Respiratory effects of opioid full and partial agonists in rhesus monkeys. J. Pharmacol. Exp. Ther. 277: 462-472, 1996. MACDONALD, F. C., GOUGH, K. J., NICOLL, R. A. AND DOW, R. J.: Psychomotor effects of ketorolac in comparison with buprenorphine and diclofenac. Br. J. Clin. Pharmacol. 27: 453-459, 1989. MANNER, T., KANTO, J. AND SALONEN, M.: Simple devices in differentiating the effects of buprenorphine and fentanyl in healthy volunteers. Eur. J. Clin. Pharmacol. 31: 673-676, 1987. MARTIN, W. R., EADES, C. G., THOMPSON, J. A., HUPPLER, R. E. AND GILBERT, P. E.: The effects of morphine- and nalorphine-like drugs in the nondependent and morphine-dependent chronic spinal dog. J. Pharmacol. Exp. Ther. 197: 517532, 1976. MARTIN, W. R., SLOAN, J. W., SAPIRA, J. D. AND JASINSKI, D. R.: Physiologic, subjective, and behavioral effects of amphetamine, methamphetamine, ephedrine, phenmetrazine, and methylphenidate in man. Clin. Pharmacol. Ther. 12: 245-258, 1971. MATHER, L. E.: Opioid analgesics. In Anesthesia, 2nd ed., ed. by W. S. Nimmo, D. J. Rowbotham and G. Smith, eds., pp. 132-165, Blackwell Scientific Publications, London, 1994. MAUNUKSELA, E. L., KORPELA, R. AND OLKKOLA, K. T.: Comparison of buprenorphine with morphine in the treatment of postoperative pain in children. Anesth. Analg. 67: 233-239, 1988. MELLO, N. K., LUKAS, S. E., BREE, M. P. AND MENDELSON, J. H.: Progressive ratio performance maintained by buprenorphine, heroin and methadone in macaque monkeys. Drug Alcohol Depend. 21: 81-97, 1988. MELLO, N. K., MENDELSON, J. H. AND KUEHNLE, J. C.: Buprenorphine effects on human self-administration: An operant analysis. J. Pharmacol. Exp. Ther. 223: 30-39, 1982. MENDELSON, J., JONES, R. T., FERNANDEZ, I., WELM, S., MELBY, A. K. AND BAGGOTT, M. J.: Buprenorphine and naloxone interactions in opiate-dependent volunteers. Clin. Pharmacol. Ther. 60: 105-114, 1996. NEGUS, S. S., PICKER, M. J. AND DYKSTRA, L. A.: Interactions between mu and kappa opioid agonists in the rat drug discrimination procedure. Psychopharmacology 102: 465-473, 1990. NEGUS, S. S., PICKER, M. J. AND DYKSTRA, L. A.: Interactions between the discriminative stimulus effects of mu and kappa opioid agonists in the squirrel monkey. J. Pharmacol. Exp. Ther. 256: 149-158, 1991. NOREN, R. L.: Opiate pharmacology. In Handbook of Critical Care Pain Management, ed. by R. J. Hammill and J. C. Rowlingson, pp. 117-141, McGrawHill, Inc., New York, 1994. NUOTTO, E. J. AND KORTTILA, K.: Evaluation of a new computerized psychomotor test battery: effects of alcohol. Pharmacol. Toxicol. 68: 360-365, 1991. OUELLETTE, R. 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