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Agents were classified into 5 comparison groups based on the index antidepressant claim: Low-Dose Target-Dose High-Dose TCAs tricyclic antidepressants Drug Range mg ; Range mg ; Range mg ; SSRIs selective serotonin reuptake inhibitors; fluoxetine, SSRI: fluvoxamine, sertraline, paroxetine, citalopram, escitalopram Citalopram 20 20-40 40 SNRIs serotonin norepinephrine reuptake inhibitors; venlafaxEscitalopram 10 ine IR, venlafaxine XR ; , bupropion; or Fluoxetine 20 20-40 40 Paroxetine 20 20-40 40 "Other antidepressant" i.e., mirtazapine, nefazadone, trazodone, Sertraline 50 50-150 150 isocarboxazid, maprotiline, phenelzine sulfate, and tranylcyFluvoxamine maleate 50 50-100 100 promine sulfate ; . SNRI: Medication use in this analysis was based on intent to treat. Venlafaxine IR or XR 37.5-74 75-150 151 + That is, if a patient initiated on an SSRI and switched or Bupropion: augmented using another drug class, their adherence rate and Bupropion hydrochloride 150 150-300 300 costs would be associated with the patient's initiation on SSRIs. Wellbutrin SR ; Bupropion hydrochloride tablet ; 200 200-300 300 No minimum duration period on the initiating drug was Other: required in order to avoid biasing the sample toward adherent Mirtazapine 15 15-30 30 patients. Overall, 12.6% of patients switched or augmented Nefazodone hydrochloride 200 300-400 400 sometime during the 6-month follow-up. Isocarboxazid 20 20-40 40 Index dosage level. Daily dose was calculated for the index Maprotiline hydrochloride 75 75-150 150 Phenelzine sulfate 45 45-60 60 medication based on the number of pills, strength, and days Tranylcypromine sulfate 30 supplied. Daily doses were then defined as low, target, or high Trazodone hydrochloride 150 150-400 400 based on the dosage ranges specified in the product insert PI ; TCA: for each drug see Table 3 ; . For example, "target dose" was Amitriptyline hydrochloride 75 75-150 150 defined as 20-40 mg per day for fluoxetine and 75-150 mg per Clomipramine hydrochloride 25 25-150 150 day for venlafaxine. Desipramine hydrochloride 100 100-200 200 Doxepin hydrochloride 75 75-150 150 Patient characteristics. Patient characteristics were based Imipramine hydrochloride 50 50-100 100 on data available at the time of the index medication claim, Imipramine pamoate 50 50-100 100 including age, gender, geographic region Northeast, North Nortriptyline hydrochloride 25 25-100 100 Central, South, or West ; , insurance plan type capitated vs. nonProtriptyline hydrochloride 45 45-100 100 Trimipramine maleate 75 75-150 150 capitated ; , and a proxy for household socioeconomic status salary * Adapted from Physicians' Desk Reference [online]. Available at: vs. hourly pay ; . Insurance plan types defined as capitated included thomsonhc hcs librarian. Accessed July 17, 2005. health maintenance organizations and point of service POS ; with IR immediate release; XR extended release; SNRI serotonin norepinephrine capitation. Noncapitated health plans included PPOs, basic major reuptake inhibitor; SR sustained release; SSRI selective serotonin reuptake medical, comprehensive, and noncapitated POS. inhibitor; TCA tricyclic antidepressant. Clinical characteristics. Comorbid anxiety and bipolar disorders were measured in the preperiods and postperiods using individual ICD-9 codes 300.0x for anxiety disorder and diagnosis and filled prescription was 7 days median 4 days ; . 296.4x, 296.5x, 296.6x, for bipolar disorder ; . Chronic The subset of overall expenditures that were depression-related disease was assessed by using inpatient and outpatient diagnoses also was assessed. to calculate the Charlson Comorbidity Index Score CCI ; .16 An Encounter records for patients in some plans are based on indicator variable identifying patients receiving any mental capitated payment records, and the payment field is rarely health specialty care any billed contact encounter coded with a populated. To address this issue, a payment rate was assigned to psychiatrist, mental health and chemical dependency treatment each procedure code based on a regionally adjusted mean pay- facility, psychologist, or psychiatric nurse ; during the study ment amount for that procedure from all Marketscan fee-for- period was also included. service claims occurring in that year. All actual and proxy payments were then adjusted to 2004 dollars using the Statistical Analysis Consumer Price Index for all Urban Consumers CPI-U ; .15 Univariate analyses, including t tests and chi-square tests, were Measures of index antidepressant class, dosage level, patient used to analyze patient and clinical characteristics by initiating characteristics, and clinical characteristics were used as treatment groups. Multivariate regression models were used independent variables. to evaluate differences across outcomes of interest: adherence to Index medication. Patients initiated on any of the following HEDIS guidelines and economic impact of adherence to HEDIS.

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This report has thus far been published in hard-copy format only. However, owing to the additional information provided in this 2005 edition e.g. registration status of medicines by country ; certain parts of the report are available only the CDROM attached to the inside back cover. A web site is also currently under development, which will house a searchable version of this report, as well as in-country registration information that will be regularly updated. It will also contain direct links to other relevant on-line resources and tools.

In Lagos, students from Bariga Comprehensive High School paint a mural for a competition held by Action Health, Inc., a Nigerian organization that promotes reproductive health and youth development. The competition, whose theme was "Mirror on the Wall, " reflects Action Health's mission to provide young people with the information, education, skills and services they need to gain greater power over their lives.
21. Nahass GT. Antiphospholipid antibodies and the antiphospholipid antibody syndrome. J Acad Dermatol. 1997; 36: 149-71. Goldberg SN, Conti-Kelly AM, Greco T. A family study of anticardiolipin antibodies and associated clinical conditions. J Med. 1995; 99: 473-9. Sebastiani GD, Galeazzi M, Morozzi G, Marcolongo R. The immunogenetics of the antiphospholipid syndrome, anticardiolipin antibodies, and lupus anticoagulant. Semin Arthritis Rheum. 1996; 25: 414-20. McNeil HP Chesterman CN, Krilis AS. Immunology and , clinical importance of antiphospholipid antibodies. Adv Immunol. 1991; 49: 193-280. Petri M. Pathogenesis and treatment of the antiphospholipid antibody syndrome. Med Clin North Am. 1997; 81: 151-77. Salmon JE, Girrardi G. The role of complement in the antiphospholipid syndrome. Curr Dir Autoimmun. 2004; 7: 133-48. Simantov R, LaSala JM, Lo SK, Gharavi AE, Sammaritano LR, Salmon JE, et al. Activation of cultured vascular endothelial cells by antiphospholipid antibodies. J Clin Invest. 1995; 96: 2211-9. Carmo-Pereira S, Bertolaccini ML, Escudero-Contreras A. Value of IgA anticariolipin and anti-beta 2-glycoprotein I antibody testing in patients with pregnancy morbidity. Ann Rheum Dis. 2003; 62: 540-3. Gezer S. Antiphospholipid Syndrome. Dis Mon. 2003; 49: 696-741. Cuadrado MJ, Hughes GRV Hughes antiphospholipid ; . syndrome. Clinical features. Rheum Dis Clin North. 2001; 27: 507-24. Amigo MC, Khamashta MA. Antiphospholipid Hughes ; syndrome in systemic lupus erythematosus. Rheum Dis Clin North Am. 2000; 26: 331-48. Hanly JG. Antiphospholipid syndrome. An overview. CMAJ. 2003; 168: 1675-82. Mulinari-Brenner FA, Pizzol ASD, Manfrinato LC, Santamaria JR, Gemelli JJ. An Brs Dermatol. 2000; 75: 219-24. Levine JS, Branch DW Rauch J. The antiphospholipid , syndrome. N Engl J Med. 2002; 346: 752-63. Digenes MJN, Digenes PCN, Carneiro RMM, et al. Cutaneous manifestations associated with antiphospholipid antibodies. Int J Dermatol. 2004; 43: 632-7. Alegre VA, Winkelmann RK, Gastineau DA. Cutaneous thrombosis, cerebrovascular thrombosis and lupus anticoagulant- the Sneddon syndrome. Report of 10 cases. Int J Dermatol. 1990; 29: 45-9. Levine SR, Langer SL, Albers JW Welch KMA. Sneddon's , syndrome: an antiphospholipid antibody syndrome?. Neurology. 1988; 38: 798-800. Gantcheva M. Dermatologic aspects of antiphospholipid syndrome. Int J Dermatol. 1998; 37: 173-80. Galli M. Antiphospholipid syndrome: association between laboratory tests and clinical practice. Pathophysiol Haemost Thromb. 2004; 33: 249-55. Greaves M. Antiphospholipid antibodies and thrombosis. Lancet. 1999; 353: 1348-53. Amengual O, Atsumi T, Koike T. Antiprothrombin antibodies and the diagnosis of antiphospholipid syndrome. Clin Immun. 2004; 112: 144-9 and treprostinil.

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Sepsis or pneumonia occurred in nine courses 18% ; . Fever of unknown origin was seen in four courses 8% ; . Minor infections were noted in six courses 12% ; . Three patients developed evidence of cumulative myelotoxicity with 2-CDA. The first patient achieved a PR of her large cell lymphoma after two courses. The platelet count at the start of treatment was 150, 000 pL, and decreased to 82, 000 after the first course. On day 35 of the second course the platelet count was 35, OOO pL. She was not given further therapy; 2 months later the platelet count was 251, OOO pL. The second patient had MF and received three courses of 2CDA. The platelet count before 2-CDA was 184, 000 pL and decreased after two courses to 74, 0001pL. He had a minor response and 2-CDA was held. By day 110 the platelet count had recovered to 107, 000 pL and he received a third course of 2-CDA for only 4 days, with a subsequent decrease of the platelet count to 59, OoO pL; because he had no further response he did not receive more 2-CDA. The third patient had LGL diagnosed on splenectomy. He received three courses of 2-CDA. The platelet count before 2-CDA was 209, 00O pL and after 2 courses was reduced to 93, OOO pL. He received a third course of 2-CDA at full dose and had a platelet nadir of 37, OoO pL on day 66. The patient was otherwise in complete remission. He had a slow rise in the platelet count, which finally increased above 100, OOO pL 19 months after the third course of 2-CDA. He continues in complete remission 54 months from the initiation of treatment. Four patients died during the first course of 2-CDA. Two patients with leukemia died of disseminated Candida infection. Both had received prior fludarabine. The only patient with a Zubrod performance status of 3 died on day 16 of the treatment. The cause of death was uncertain but likely related to progressive disease. One patient with lymphoma died at 4 weeks with progressive disease. Nonhematologic toxicity was minimal with 2-CDA. One patient had nausea and vomiting with both courses of 2CDA. One patient complained of increasing fatigue. One patient noted numbness in the feet.

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This research was partly funded by a research grant pb 96-0413 ; of the spanish government cicyt.

Outcome of total knee arthroplasty. A study comparing questionnaires, clinical evaluation and radiological assessment * Evans, A; Hussain, F; Oni, O Wallaby 1 : 1 years follow-up of 500 PCL sparing knee prosthesis * Nizard, R; Masse, Y; Witvoet, J; Nordin, J Y Soft-tissue balancing in fixed valgus TKA: methods of achieving concave side releases * Boldt, J; Keblis, P; Briaird, J L Total knee arthroplasty in osteoarthritis: a long-term results of 943 knees * Joshi, A; Gurdev, G Cemented vs HAC AGC TKR. A prospective randomised controlled trial * Butler-Manuel, A; Apthorp, H; Rogers, A Results after 7 years [6-8] of follow-up of a total knee prosthesis using a third condyle for posterior stabilization * Migaud, H; Becquet, E; Chantelot, C; Ala Eddine, T; Gougeon, F; Duquennoy, A Total knee arthroplasty in young: results at minimum of 10 years * Gurdey, G; Joshi, A Early outcomes and complications of the PFC sigma knee replacement * Patsalides, C; Tobin, S; Bobic, V and triazolam.

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Transfused persons EV: Transfuse BT: People Transfusion BT: Blood treatments RT: Anemia SN: Transfusion refers to increased HIV disease progression in HIV patients who receive blood trans-fusions. Refer to Blood to blood transmission for contracting HIV by a blood transfusion. Transgendered persons EV: Transgendriste BT: People NT: Transexuals Transvestites Translation EV: Traduction [gntique] BT: Molecular genetic processes RT: tRNA Transmission EV: Transmission [VIH] NT: Blood to blood transmission Occupational transmission Sexual transmission Vertical transmission Zoonotic transmission RT: Health promotion High risk behaviours SN: CATIE Thesaurus has moderate coverage of HIV disease transmission issues. CATIE terminology includes: Blood to blood transmission; Sexual transmission Heterosexual transmission, Homosexual transmission Vertical transmission Perinatal transmission, Postnatal transmission, and Prenatal transmission and Zoonotic transmission. IMPORTANT: PLEASE READ Do you have high blood pressure? Have you ever had to stop taking this or any other medication because of an allergy or other problems? Are you taking any other migraine 5-HT1 agonist medications such as sumatriptan succinate, naratriptan hydrochloride, rizatriptan benzoate, almotriptan malate or migraine medications containing ergotamine, dihydroergotamine, or methysergide? Are you taking any medications for depression such as selective serotonin reuptake inhibitors SSRI's ; , for example, fluoxetine hydrochloride, sertraline hydrochloride, fluvoxamine maleate, paroxetine hydrochloride, etc., or serotonin norepinephrine reuptake inhibitors SNRIs ; , for example, venlafaxine hydrochloride, or monoamine oxidase inhibitors MAOIs ; , for example, phenelzine sulfate, tranylcypromine sulfate or moclobemide? Have you ever experienced numbness on one side of your body when you have a headache? Have you ever had epilepsy or seizures? Have you ever had liver disease? Are you over 65 years of age? Is this headache different from your usual migraine attacks? Are you taking cimetidine for treatment of indigestion or stomach ulcers ; or a member of the quinolone family of antibiotics for example ciprofloxacin ; ? antibiotics from the quinolone family for example ciprofloxacin and trifluoperazine. Krupitsky E, Grinenko A, Karandashova G, Berkaliev T, Moshkov K, Borodkin, Y. Metabolism of biogenic amines induced by alcoholism narcopsychotherapy with ketamine administration. Biogenic Amines 7 1990 ; : 577-582 Krupitsky E, Grinenko A, Berkaliev T, Paley A, Petrov V, Moshkov K, Borodkin, Y. The combination of psychedelic and aversive approaches in alcoholism treatment: the affective contra-attribution method. Alcoholism Treatment Quarterly 9 1992 ; : 99-105. Krupitsky E, Grinenko A. Ketamine Psychedelic Therapy KPT ; : A Review of the results of ten years of research. Journal of Psychoactive Drugs29 1997 ; 2: 165-183. Krupitsky E, Burakov A, Romanova T, Strassman R, Grinenko A. Ketamine-Assisted Psychotherapy KPT ; of Heroin Addiction: Immediate Effects and Six Months Follow-up. Bulletin of the Multidisciplinary Association for Psychedelic Studies9 1999 ; 4: 21-26. : maps news-letters v09n4 09421kru Krupitsky E. Ketamine Psychedelic Therapy in the Treatment of Heroin Addiction v04 Research Protocol : maps research ketamine 04Kheroin Krupnick J, Sotsky S, Simmens S, Moyer J, Elkin I, Watkins J, Pilkonis P. The role of the therapeutic alliance in psychotherapy and pharmacotherapy outcome: findings in the National Institute of Mental Health Treatment of Depression Collaborative Research Program.Journal of Consulting and Clinical Psychology64 June 1996 ; 3: 532-9. Kunz R, Oxman A. The unpredictability paradox: Review of empirical comparisons of randomized and non-randomized clinical trials. British Medical Journal 317 1998 ; : 11851190. Kurland A, Unger S, Shaffer J, Savage C. Psychedelic Therapy Utilizing LSD in the Treatment of the Alcoholic Patient: A Preliminary Report. American Journal of Psychiatry 123 1967 ; 12: 1202-1209. Kurland A, Savage C, Pahnke W, Grof S, Olsson J. LSD in the Treatment of Alcoholics. Pharmakopsychiatrie Neuro-Psychopharmacologie4 1971 ; 2: 84. Kurland A. Psychiatric Aspects of Opiate Dependence.West Palm Beach: CRC Press, 1978. Kurland A. LSD in the supportive care of the terminally ill cancer patient. Journal of 400.

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The mean value in the upper layer of 86 mg DHTDMAC kg corresponds to 36 mg DODMAC kg. With a sludge application of 5 t instead of 23 t DODMAC concentration of 7.8 mg kg would be expected. Further measurements are reported in ECETOC, 1993 original literature not available ; : In 42 fields that received their last sludge application prior to 1987 the year of sampling ; , DHTDMAC concentrations from 2 mg kg to 37 mg kg mean 9.5 mg kg ; were detected. 95 and trihexyphenidyl.

Selecting the patient: tranylcypromine should be used for the symptomatic treatment of moderate to severe depression. Tranylcypromine warrants further study in narcolepsy and trimethobenzamide. This suggests that the strength of these metal complexes is similar because complexation of the metal ion by the ligand in solution occurs at the phosphonate oxygen atoms for both ligands. In conclusion, it has been reported in literature that the potency of APD is 100 times that of HEDP i.e. to deliver radionuclei of interest to bone surface ; . It has been demonstrated in this work that the mode of complex formation for both HEDP and APD is the same. Thus the potency of APD is not related to the strength of the APD complexes, but might be related to interaction of the R-group in APD with the bone environment and tranylcypromine.

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